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IIT Ropar professor bags AAPI Young Investigator Award 2018

By   /  January 8, 2018  /  Comments Off on IIT Ropar professor bags AAPI Young Investigator Award 2018

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Ropar, 8 January 2018: Dr. Durba Pal, Assistant Professor, Biomedical Engineering, IIT Ropar has been awarded 1st prize in the prestigious AAPI Young Investigator Award 2018 for her work in ‘miR-200b, a potential therapeutic target for diabetic wound healing’. Dr. Durba Pal received citation and cash award of Rs. 1 Lakh for presenting poster followed by oral lecture at AAPI Global Healthcare Summit 2017 which was held at Calcutta Medical College, Kolkata in December 2017.

The Association of American Physicians of Indian Origin (AAPI) hosted its 11th annual Global Healthcare Summit (GHS) in Kolkata during December 2017. The summit was organized by the association in collaboration with the Ministry of Overseas Indian Affairs, the Ministry of Health and the government of West Bengal. More than 50 renowned speakers from the United States, the US and other parts of the world, as well as industry leaders were present at the event. With over 200 physicians from the United States, the summit was attended by nearly 1,000 delegates from around the world.

The competition was held among the Young Physicians Researchers based on the innovative research work and its significance in human mankind.

Speaking about the achievement, Dr. Durba Pal, Assistant Professor, Biomedical Engineering, IIT Ropar said “It is an honour to have been awarded 1st prize from among the best of young physicians researchers. This award provides support for young researchers early in their career to pursue path-breaking research with a societal impact. My research work shows that a specific non-coding RNA, microRNA 200b, could be used as a potential therapeutic target for diabetic wound healing.”

Explaining about the work, Prof. Durba Pal said, “High blood glucose level in diabetic patient is detrimental for wound healing and our work showed that following injury a transient down regulation of miR200b in endothelial cells favours rapid wound healing. High expression of miR200b in diabetic wound endothelial cells cause for its dysfunction leading to the impairment of wound healing. We have shown that diabetic wound can be healed by delivery of anti-miR200b LNA at the wound edge. This is the first evidence showing miR200b as a potential therapeutic target for diabetic wound healing.

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